Carlos H. Nitta - Concepedia

Author

Carlos H. Nitta

University of New Mexico

Also Known As

C NITTA, C. H. Nitta, C. Nitta, C.H. Nitta, Carlos H Nitta, Carlos H. Nitta, Carlos Hiroshi Nitta, Carlos Nitta

Publications

354

Citations

H-Index

Concepts

All Affiliations

University of New Mexico

About

Carlos H. Nitta is an author at University of New Mexico specializing in medicine, physiology, and vascular biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	5	5	197
2	Public Health	3	3	130
3	Cell Biology	3	3	104
4	Biochemistry	2	2	73
5	Immunology	2	2	88

Carlos H. Nitta

Publications

348

Citations

H-Index

Page 1

	Year	Citations
NFATc3 is required for chronic hypoxia-induced pulmonary hypertension in adult and neonatal mice Ryann Bierer, Carlos H. Nitta, Jessica Friedman, American Journal of Physiology-Lung Cellular and Molecular Physiology	2011	68
Role of ASIC1 in the development of chronic hypoxia-induced pulmonary hypertension Carlos H. Nitta, David A. Osmond, Lindsay M. Herbert, American Journal of Physiology-Heart and Circulatory Physiology Acute Lung InjuryHypertensionInflammatory Lung DiseaseLung InflammationImmunology	2013	57
NFAT is required for spontaneous pulmonary hypertension in superoxide dismutase 1 knockout mice Juan Manuel Ramiro‐Diaz, Carlos H. Nitta, Levi D. Maston, American Journal of Physiology-Lung Cellular and Molecular Physiology Lung InflammationPulmonary HypertensionRedox BiologyOxidative StressInflammation	2013	42
Endothelin-1 contributes to increased NFATc3 activation by chronic hypoxia in pulmonary arteries Sergio de Frutos, Juan Manuel Ramiro Diaz, Carlos H. Nitta, American Journal of Physiology-Cell Physiology	2011	39
Intermittent hypoxia-induced increases in reactive oxygen species activate NFATc3 increasing endothelin-1 vasoconstrictor reactivity Jessica Friedman, Carlos H. Nitta, K. M. Henderson, Vascular Pharmacology InflammationReactive Oxygen SpeciesMedicinePhysiologyHypoxia (Medicine)	2013	36
Regulation of soluble guanylyl cyclase-α1 expression in chronic hypoxia-induced pulmonary hypertension: role of NFATc3 and HuR Sergio de Frutos, Carlos H. Nitta, Elizabeth Caldwell, American Journal of Physiology-Lung Cellular and Molecular Physiology HypertensionLung InflammationSgc Gene ExpressionPulmonary HypertensionOxidative Stress	2009	31
NFATc3 contributes to intermittent hypoxia-induced arterial remodeling in mice Sergio de Frutos, Elizabeth Caldwell, Carlos H. Nitta, American Journal of Physiology-Heart and Circulatory Physiology HypertensionImmunologySelective Endothelin ReceptorOxidative StressSleep-related Breathing Disorder	2010	31
PICK1/calcineurin suppress ASIC1-mediated Ca<sup>2+</sup> entry in rat pulmonary arterial smooth muscle cells Lindsay M. Herbert, Carlos H. Nitta, Tracylyn R. Yellowhair, American Journal of Physiology-Cell Physiology	2016	17
Aquaporin-2 Promoter Is Synergistically Regulated by Nitric Oxide and Nuclear Factor of Activated T Cells María F. Albertoni Borghese, Layne M. Bettini, Carlos H. Nitta, Nephron Extra	2011	14
Autophagy response to acute high-intensity interval training and moderate-intensity continuous training is dissimilar in skeletal muscle and peripheral blood mononuclear cells and is influenced by sex Kurt A. Escobar, Anna M. Welch, Andrew Wells, Human Nutrition & Metabolism	2020	13

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