Joseph Gera - Concepedia

Author

Joseph Gera

University of California, Los Angeles ORCID

Also Known As

J F Gera, J Gera, J. Gera, Joseph F. Gera, Joseph Gera, Yilun Liu, de Jesus, Jason

155

Publications

4.3K

Citations

H-Index

Concepts

All Affiliations

University of California, Los Angeles VA Greater Los Angeles Healthcare System UCLA Jonsson Comprehensive Cancer Center UCLA Medical Center Ronald Reagan UCLA Medical Center

About

Joseph Gera is an author at University of California, Los Angeles specializing in cell biology, medicine, and cell signaling.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	21	28	1.9K
2	Cell Biology	21	27	1.8K
3	Natural Sciences	9	9	277
4	Biochemistry	3	3	103
5	Engineering	1	1	19

Joseph Gera

Publications

4.2K

Citations

H-Index

Page 1

	Year	Citations
Mammalian target of rapamycin inhibitors activate the AKT kinase in multiple myeloma cells by up-regulating the insulin-like growth factor receptor/insulin receptor substrate-1/phosphatidylinositol 3-kinase cascade Yijiang Shi, Huajun Yan, Patrick Frost, Molecular Cancer Therapeutics	2005	369
AKT Activity Determines Sensitivity to Mammalian Target of Rapamycin (mTOR) Inhibitors by Regulating Cyclin D1 and c-myc Expression Joseph Gera, Ingo K. Mellinghoff, Yijiang Shi, Journal of Biological Chemistry Akt ActivityC-myc ExpressionCyclin D1ImmunologyMammalian Target	2004	329
Oncogenic EGFR Signaling Activates an mTORC2–NF-κB Pathway That Promotes Chemotherapy Resistance Kazuhiro Tanaka, Ivan Babić, David A. Nathanson, Cancer Discovery	2011	278
mTORC2 Activity Is Elevated in Gliomas and Promotes Growth and Cell Motility via Overexpression of Rictor Janine Masri, Andrew Bernath, Jheralyn Martin, Cancer Research	2007	254
Enhanced sensitivity of multiple myeloma cells containing PTEN mutations to CCI-779. Yijiang Shi, Joseph Gera, Liping Hu, PubMed Hematological MalignancyTumor BiologyPten FunctionMedicineImmunology	2002	234
In vivo antitumor effects of the mTOR inhibitor CCI-779 against human multiple myeloma cells in a xenograft model Patrick Frost, Farhad Moatamed, Bao Hoang, Blood	2004	188
Tristetraprolin regulates Cyclin D1 and c-Myc mRNA stability in response to rapamycin in an Akt-dependent manner via p38 MAPK signaling M Marderosian, Asha Sharma, Alexander Funk, Oncogene Signal TransductionSignaling PathwayP38 Mapk SignalingMolecular PathwayC-myc Mrna Stability	2006	155
Cyclin D1 and c-myc Internal Ribosome Entry Site (IRES)-dependent Translation Is Regulated by AKT Activity and Enhanced by Rapamycin through a p38 MAPK- and ERK-dependent Pathway Yijiang Shi, Anushree Sharma, Hong Wu, Journal of Biological Chemistry P38 Mapk-Akt ActivitySignal TransductionRapamycin ProteinSignaling Pathway	2005	154
RETRACTED: Downstream effectors of oncogenic ras in multiple myeloma cells Liping Hu, Yijiang Shi, Jung-hsin Hsu, Blood	2003	143
Heterogeneous Nuclear Ribonucleoprotein A1 Regulates Cyclin D1 and c-myc Internal Ribosome Entry Site Function through Akt Signaling Oak D. Jo, Jheralyn Martin, Andrew Bernath, Journal of Biological Chemistry	2008	125

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