Jorge J. Palop - Concepedia

Author

Jorge J. Palop

Also Known As

Catherine Priest, J J Palop, J.J Palop, J.J. Palop, Jorge J Palop, Jorge J. Palop, Jorge Palop, Jorge J. Palop, Palop, Jorge J.

Publications

15.4K

Citations

H-Index

Concepts

All Affiliations

Gladstone Institutes University of California, San Francisco University of California San Diego Universitat de València University of California, Los Angeles

About

Jorge J. Palop is an author at Gladstone Institutes specializing in medicine, neuroscience, and neurology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	19	22	6.4K
2	Social Sciences	14	14	5.1K
3	Cell Biology	7	7	1.1K
4	Engineering	1	1	249
5	Health Sciences	1	1	11

Jorge J. Palop

Publications

10.9K

Citations

H-Index

Page 1

	Year	Citations
Reducing Endogenous Tau Ameliorates Amyloid ß-Induced Deficits in an Alzheimer's Disease Mouse Model Erik D. Roberson, Kimberly Scearce‐Levie, Jorge J. Palop, Science	2007	1.9K
Aberrant Excitatory Neuronal Activity and Compensatory Remodeling of Inhibitory Hippocampal Circuits in Mouse Models of Alzheimer's Disease Jorge J. Palop, Jeannie Chin, Erik D. Roberson, Neuron Compensatory RemodelingAlzheimer's DiseaseBrain FunctionMedicineDementia	2007	1.6K
Inhibitory Interneuron Deficit Links Altered Network Activity and Cognitive Dysfunction in Alzheimer Model Laure Verret, Edward O. Mann, Giao B. Hang, Cell Alzheimer ModelNeuropsychologyAlzheimer's DiseaseCognitive ScienceBrain Function	2012	1.2K
Amyloid-β/Fyn–Induced Synaptic, Network, and Cognitive Impairments Depend on Tau Levels in Multiple Mouse Models of Alzheimer's Disease Erik D. Roberson, Brian Halabisky, Jong W. Yoo, Journal of Neuroscience Synaptic TransmissionNeurochemical BiomarkersMultiple Mouse ModelsTau LevelsSynaptic Signaling	2011	640
Levetiracetam suppresses neuronal network dysfunction and reverses synaptic and cognitive deficits in an Alzheimer’s disease model Pascal E. Sanchez, Lei Zhu, Laure Verret, Proceedings of the National Academy of Sciences	2012	636
Accelerating Amyloid-β Fibrillization Reduces Oligomer Levels and Functional Deficits in Alzheimer Disease Mouse Models Irene H. Cheng, Kimberly Scearce‐Levie, Justin Legleiter, Journal of Biological Chemistry Neurodegenerative DiseasesArctic MutationAlzheimer's DiseaseAssembly StateDementia	2007	406
Neuronal depletion of calcium-dependent proteins in the dentate gyrus is tightly linked to Alzheimer's disease-related cognitive deficits Jorge J. Palop, Brian W. Jones, Lisa Kekonius, Proceedings of the National Academy of Sciences	2003	398
Distinct Roles of GABAergic Interneurons in the Regulation of Striatal Output Pathways Aryn H. Gittis, Alexandra Nelson, Myo T. Thwin, Journal of Neuroscience Striatal Output PathwaysSynaptic TransmissionStriatal InterneuronsNeurotransmissionSocial Sciences	2010	377
Phospholipase A2 reduction ameliorates cognitive deficits in a mouse model of Alzheimer's disease Rene O. Sanchez-Mejia, John W. Newman, Sandy Toh, Nature Neuroscience Alzheimer's DiseaseMolecular NeuroscienceMedicineDementiaPhospholipase A2 Reduction	2008	366
Transsynaptic Progression of Amyloid-β-Induced Neuronal Dysfunction within the Entorhinal-Hippocampal Network Julie A. Harris, Nino Devidze, Laure Verret, Neuron Synaptic PlasticityNeurodegenerative DiseasesSynaptic NeuroscienceSynaptic TransmissionNeurology	2010	325

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