William E. Balch - Concepedia

Author

William E. Balch

Also Known As

Balch, W. E., Balch, William, Kellner, Wendy A., Pilewski, Joseph M, Skach, William R., W BALCH, W E Balch, W. Balch, W. E. BALCH, W. E. Balch

327

Publications

33K

Citations

H-Index

151

Concepts

All Affiliations

Scripps Research Institute Torrey Pines Institute For Molecular Studies Scripps Institution of Oceanography Scripps (United States)University of California San Diego

About

William E. Balch is an author at Scripps Research Institute specializing in medicine, natural sciences, and cell biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	64	91	16.5K
2	Natural Sciences	51	76	11.7K
3	Engineering	5	5	513
4	Education	1	1	44
5	Public Health	1	1	27

William E. Balch

160

Publications

23.8K

Citations

H-Index

Page 1

	Year	Citations
Monocyte-derived alveolar macrophages drive lung fibrosis and persist in the lung over the life span Alexander V. Misharin, Luisa Morales‐Nebreda, Paul A. Reyfman, The Journal of Experimental Medicine Acute Lung InjuryMonocyte-derived Alveolar MacrophagesInflammatory Lung DiseaseAdvanced Lung DiseaseLung Inflammation	2017	1.1K
Functional Amyloid Formation within Mammalian Tissue Douglas M. Fowler, Atanas V. Koulov, Christelle Alory-Jost, PLoS Biology Protein FunctionAlzheimer's DiseaseProtein AssemblyBiochemistryProtein Folding	2005	808
Reconstitution of the transport of protein between successive compartments of the golgi measured by the coupled incorporation of N-acetylglucosamine William E. Balch, William G. Dunphy, William A. Braell, Cell Golgi ApparatusProtein SecretionBiochemistryProtein FoldingNatural Sciences	1984	695
Hsp90 Cochaperone Aha1 Downregulation Rescues Misfolding of CFTR in Cystic Fibrosis Xiaodong Wang, John D. Venable, Paul LaPointe, Cell FibrosisSystems BiologyPathologyPulmonary FibrosisGene Expression	2006	595
From CFTR biology toward combinatorial pharmacotherapy: expanded classification of cystic fibrosis mutations Radu G. Avramescu, Annette Chiang, Scott A. Houck, Molecular Biology of the Cell PathologyCombinatorial PharmacotherapySynaptic SignalingCellular PhysiologyMolecular Pharmacology	2016	582
Chemical chaperones increase the cellular activity of N370S β-glucosidase: A therapeutic strategy for Gaucher disease Anu R. Sawkar, Wei‐Chieh Cheng, Ernest Beutler, Proceedings of the National Academy of Sciences	2002	501
GTP-binding mutants of rab1 and rab2 are potent inhibitors of vesicular transport from the endoplasmic reticulum to the Golgi complex. E J Tisdale, J R Bourne, Roya Khosravi‐Far, The Journal of Cell Biology Viral ReplicationMolecular BiologyRas-related Rab ProteinsViral Structural ProteinCellular Physiology	1992	500
A Di-Acidic Signal Required for Selective Export from the Endoplasmic Reticulum Noriyuki Nishimura, William E. Balch Science Protein SecretionCytoskeletonCellular PhysiologyMembrane ProteinsEndocytic Pathway	1997	488
The Biological and Chemical Basis for Tissue-Selective Amyloid Disease Yoshiki Sekijima, R. Luke Wiseman, Jeanne Matteson, Cell Alzheimer's DiseaseTissue-selective Amyloid DiseaseBiochemistryDegenerative PathologyProtein Misfolding	2005	486
Rab1 Recruitment of p115 into a cis-SNARE Complex: Programming Budding COPII Vesicles for Fusion Bernard B. Allan, Bryan D. Moyer, William E. Balch Science	2000	450

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