FGF23 induces left ventricular hypertrophy

Christian Faul, Ansel P. Amaral, Behzad N. Oskouei, +32

Journal of Clinical Investigation

HypertensionHeart FailurePathologyCardiovascular FunctionMetabolic Syndrome +16

The actin cytoskeleton of kidney podocytes is a direct target of the antiproteinuric effect of cyclosporine A

Christian Faul, Mary Donnelly, Sandra Merscher, +9

Nature Medicine

UrologyOsmoregulationKidney PodocytesCell SignalingPhysiology +10

TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal function

Jochen Reiser, Krishna R. Polu, Clemens Möller, +15

Nature Genetics

UrologySignal TransductionMolecular PhysiologyNormal Renal FunctionRenal Function +8

Gain-of-function RAF1 mutations cause Noonan and LEOPARD syndromes with hypertrophic cardiomyopathy

Bhaswati Pandit, Anna Sárközy, L Pennacchio, +26

Nature Genetics

CardiomyopathyMendelian DisorderGenetic DisorderGeneticsGain-of-function Raf1 Mutations +4

Induction of B7-1 in podocytes is associated with nephrotic syndrome

Jochen Reiser, Gero von Gersdorff, Martin Loos, +15

Journal of Clinical Investigation

Podocin, a raft-associated component of the glomerular slit diaphragm, interacts with CD2AP and nephrin

Karin Schwarz, Matias Simons, Jochen Reiser, +6

Journal of Clinical Investigation

Activation of Cardiac Fibroblast Growth Factor Receptor 4 Causes Left Ventricular Hypertrophy

Alexander Grabner, Ansel P. Amaral, Karla Schramm, +28

Cell Metabolism

Cardiac MuscleCardiomyopathyHeart FailurePhysiologyMedicine +3

Podocin, a raft-associated component of the glomerular slit diaphragm, interacts with CD2AP and nephrin

Karin Schwarz, Matias Simons, Jochen Reiser, +6

Journal of Clinical Investigation

Podocin ServesCytoskeletonCellular PhysiologyEndocytic PathwayRaft-associated Component +18

Synaptopodin orchestrates actin organization and cell motility via regulation of RhoA signalling

Katsuhiko Asanuma, Etsuko Yanagida-Asanuma, Christian Faul, +3

Nature Cell Biology

Actin OrganizationSignal TransductionCell InteractionCytoskeletonCell Motility +5

COQ6 mutations in human patients produce nephrotic syndrome with sensorineural deafness

Saskia F. Heeringa, Gil Chernin, Moumita Chaki, +52

Journal of Clinical Investigation