BioID identifies novel c-MYC interacting partners in cultured cells and xenograft tumors

Dharmendra Dingar, Manpreet Kalkat, Pak-Kei Chan, +11

Journal of Proteomics

Cultured CellsOncogenic AgentMedicineBioid IdentifiesXenograft Tumors +5

MYC Interacts with the G9a Histone Methyltransferase to Drive Transcriptional Repression and Tumorigenesis

William B. Tu, Yu-Jia Shiah, Corey Lourenco, +12

Cancer Cell

Histone ModificationsChromatinTranscriptional RegulationChromatin RemodelingNatural Sciences +12

MYC dephosphorylation by the PP1/PNUTS phosphatase complex regulates chromatin binding and protein stability

Dharmendra Dingar, William B. Tu, Diana Resetca, +9

Nature Communications

Effect of pressure overload-induced hypertrophy on the expression and localization of p38 MAP kinase isoforms in the mouse heart

Dharmendra Dingar, Clémence Merlen, Scott A. Grandy, +5

Cellular Signalling

Cardiac MuscleHypertensionHeart FailureSignal TransductionMolecular Physiology +11

Inhibiting MYC binding to the E-box DNA motif by ME47 decreases tumour xenograft growth

Lindsay C. Lustig, Dharmendra Dingar, William B. Tu, +7

Oncogene

Tumor BiologyChromatinOncogenic AgentMedicineMyc Binding +9

MYC Phosphorylation at Novel Regulatory Regions Suppresses Transforming Activity

Amanda R. Wasylishen, Michelle Chan‐Seng‐Yue, Christina Bros, +10

Cancer Research

Systems BiologySignal TransductionCell RegulationMyc PhosphorylationMedicine +12

Anti-apoptotic function of the E2F transcription factor 4 (E2F4)/p130, a member of retinoblastoma gene family in cardiac myocytes

Dharmendra Dingar, Filip Konečný, Jian Zou, +2

Journal of Molecular and Cellular Cardiology

Retinoblastoma Gene FamilyCell RegulationCell DeathCardiac MyocytesAnti-apoptotic Function +6

Characterization of the expression and regulation of MK5 in the murine ventricular myocardium

Dharmendra Dingar, Marie-Josée Benoît, Aida M. Mamarbachi, +6

Cellular Signalling

Cardiac MuscleCardiomyopathyMolecular PhysiologySignal TransductionPhysiology +10

MK5 haplodeficiency attenuates hypertrophy and preserves diastolic function during remodeling induced by chronic pressure overload in the mouse heart

Sherin A. Nawaito, Dharmendra Dingar, Pramod Sahadevan, +7

American Journal of Physiology-Heart and Circulatory Physiology

MK2‐Deficient Mice Are Bradycardic and Display Delayed Hypertrophic Remodeling in Response to a Chronic Increase in Afterload

Matthieu Ruiz, Maya Khairallah, Dharmendra Dingar, +19

Journal of the American Heart Association