Hannah J. Brown - Concepedia

Author

Hannah J. Brown

University of Pennsylvania ORCID

Also Known As

H Brown, H J Brown, H. Brown, Hannah Brown, Hannah J. Brown, Hannah Jean Brown, M. Tintel

Publications

2.3K

Citations

H-Index

Concepts

All Affiliations

University of Pennsylvania Institute on Aging Rush University Medical Center The Ohio State University Wexner Medical Center Santa Fe Institute

About

Hannah J. Brown is an author at University of Pennsylvania specializing in medicine, neuroscience, and neurodegenerative diseases.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	6	6	924
2	Social Sciences	4	4	777
3	Allergy	1	1	28
4	Allergy Medicine	1	1	28
5	Biochemistry	1	1	119

Hannah J. Brown

Publications

1.7K

Citations

H-Index

Page 1

	Year	Citations
Spread of α-synuclein pathology through the brain connectome is modulated by selective vulnerability and predicted by network analysis Michael X. Henderson, Eli J. Cornblath, Adam S. Darwich, Nature Neuroscience Brain ConnectomeSynaptic SignalingSocial SciencesNeurobiology Of DiseaseSynaptic Neuroscience	2019	304
A novel CDK5‐dependent pathway for regulating GSK3 activity and kinesin‐driven motility in neurons Gerardo Morfini, Györgyi Szebenyi, Hannah J. Brown, The EMBO Journal	2004	264
Patient-derived frontotemporal lobar degeneration brain extracts induce formation and spreading of TDP-43 pathology in vivo Sílvia Porta, Yan Xu, Clark R. Restrepo, Nature Communications	2018	246
Amyloid-Beta (Aβ) Plaques Promote Seeding and Spreading of Alpha-Synuclein and Tau in a Mouse Model of Lewy Body Disorders with Aβ Pathology Fares Bassil, Hannah J. Brown, Shankar Pattabhiraman, Neuron Neurodegenerative DiseasesPlaques Promote SeedingProteinopathiesDegenerative DiseaseDegenerative Pathology	2019	228
Glucocerebrosidase Activity Modulates Neuronal Susceptibility to Pathological α-Synuclein Insult Michael X. Henderson, Samantha F. Sedor, Ian McGeary, Neuron Neurodegenerative DiseasesPathological α-SynucleinNeurobiology Of DiseaseSynaptic TransmissionDegenerative Pathology	2019	142
Inhibition of Fast Axonal Transport by Pathogenic SOD1 Involves Activation of p38 MAP Kinase Gerardo Morfini, Daryl A. Bosco, Hannah J. Brown, PLoS ONE P38 MitogenP38 Map KinaseImmunologyCell DeathCytoskeleton	2013	119
α-Synuclein modulates tau spreading in mouse brains Fares Bassil, Emily S. Meymand, Hannah J. Brown, The Journal of Experimental Medicine Synaptic TransmissionMouse BrainsNeurochemical BiomarkersSimultaneous IntroductionSynaptic Signaling	2020	103
Computational modeling of tau pathology spread reveals patterns of regional vulnerability and the impact of a genetic risk factor Eli J. Cornblath, Howard L. Li, Lakshmi Changolkar, Science Advances	2021	63
GFP-Mutant Human Tau Transgenic Mice Develop Tauopathy Following CNS Injections of Alzheimer's Brain-Derived Pathological Tau or Synthetic Mutant Human Tau Fibrils Garrett S. Gibbons, Rachel A. Banks, Bumjin Kim, Journal of Neuroscience	2017	53
LRRK2 inhibition does not impart protection from α-synuclein pathology and neuron death in non-transgenic mice Michael X. Henderson, Medha Sengupta, Ian McGeary, Acta Neuropathologica Communications	2019	48

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