Mohammad B. Aljazi - Concepedia

Author

Mohammad B. Aljazi

Michigan State University ORCID

Also Known As

Mohammad Aljazi, Mohammad B Aljazi, Mohammad B. Aljazi

Publications

281

Citations

H-Index

Concepts

All Affiliations

Michigan State University Michigan Department of Agriculture and Rural Development

About

Mohammad B. Aljazi is an author at Michigan State University specializing in medicine, natural sciences, and cell biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	5	5	207
2	Natural Sciences	3	3	38
3	Cell Biology	3	3	38
4	Health Sciences	1	1	150
5	Social Sciences	1	1	19

Mohammad B. Aljazi

Publications

274

Citations

H-Index

Page 1

	Year	Citations
Liver-specific loss of Perilipin 2 alleviates diet-induced hepatic steatosis, inflammation, and fibrosis Charles P. Najt, S. Subramanian, Mohammad B. Aljazi, American Journal of Physiology-Gastrointestinal and Liver Physiology Diet-induced Hepatic SteatosisLipid PeroxidationImmunologyPathologyLiver-specific Loss	2016	150
Loss of histone methyltransferase ASH1L in the developing mouse brain causes autistic-like behaviors Yuen Gao, Natalia Duque‐Wilckens, Mohammad B. Aljazi, Communications Biology	2021	30
Kdm6b Haploinsufficiency Causes ASD/ADHD-Like Behavioral Deficits in Mice Yuen Gao, Mohammad B. Aljazi, Jin He Frontiers in Behavioral Neuroscience	2022	24
Vorinostat, a histone deacetylase inhibitor, ameliorates the sociability and cognitive memory in an Ash1L-deletion-induced ASD/ID mouse model Yuen Gao, Mohammad B. Aljazi, Yan Wu, Neuroscience Letters Brain FunctionBrain DevelopmentHistone Deacetylase InhibitorGeneticsNeurochemical Biomarkers	2021	19
Cell Signaling Coordinates Global PRC2 Recruitment and Developmental Gene Expression in Murine Embryonic Stem Cells Mohammad B. Aljazi, Yuen Gao, Yan Wu, iScience Developmental Gene ExpressionCell SpecializationEpigeneticsTranscriptional RegulationSignaling Pathway	2020	14
Histone H3K36me2-Specific Methyltransferase ASH1L Promotes MLL-AF9-Induced Leukemogenesis Mohammad B. Aljazi, Yuen Gao, Yan Wu, Frontiers in Oncology	2021	13
SMYD5 is a histone H3-specific methyltransferase mediating mono-methylation of histone H3 lysine 36 and 37 Mohammad B. Aljazi, Yuen Gao, Yan Wu, Biochemical and Biophysical Research Communications Histone ModificationsHistone H3Epigenetic ChangeGeneticsMolecular Biology	2022	13
Impaired KDM2B-mediated PRC1 recruitment to chromatin causes defective neural stem cell self-renewal and ASD/ID-like behaviors Yuen Gao, Natalia Duque‐Wilckens, Mohammad B. Aljazi, iScience ChromatinDevelopmental BiologyChromatin StructureChromatin RemodelingMedicine	2022	11

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