James W. McNamara

Hypertrophic cardiomyopathy mutations in <i>MYBPC3</i> dysregulate myosin

Christopher N. Toepfer, Hiroko Wakimoto, Amanda C. Garfinkel et al. · Science Translational Medicine · 2019 · 214 citations · Full text

Life Sciences, Genetics, Molecular Biology +13

The role of super-relaxed myosin in skeletal and cardiac muscle

James W. McNamara, Amy Li, Cristobal G. dos Remedios et al. · Biophysical Reviews · 2014 · 187 citations · Full text

Natural Sciences, Biophysics, Physiology +11

Ablation of cardiac myosin binding protein-C disrupts the super-relaxed state of myosin in murine cardiomyocytes

James W. McNamara, Amy Li, Nicola J. Smith et al. · Journal of Molecular and Cellular Cardiology · 2016 · 152 citations · Full text

Life Sciences, Cell Biology, Molecular Biology +16

MYBPC3 mutations are associated with a reduced super-relaxed state in patients with hypertrophic cardiomyopathy

James W. McNamara, Amy Li, Sean Lal et al. · PLoS ONE · 2017 · 148 citations · Full text

Medicine, Cardiology, Molecular Biology +13

Phosphoproteomics of three exercise modalities identifies canonical signaling and C18ORF25 as an AMPK substrate regulating skeletal muscle function

Ronnie Blazev, Christian S. Carl, Yaan‐Kit Ng et al. · Cell Metabolism · 2022 · 123 citations

Life Sciences, Biochemistry, Physiology +12