Virginie Buggia-Prévot

Author

Also Known As

V. Buggia-Prevot, V. Buggia-Prévot, Virginie Buggia-Prevot, Virginie Buggia-Prévot, Virginie Buggia‐Prevot, Virginie Buggia‐Prévot

Publications

1.5K

Citations

H-Index

Concepts

All Affiliations

University of Chicago The University of Texas MD Anderson Cancer Center Northwestern University University of Nebraska Medical Center University of Zurich

About

Virginie Buggia-Prévot is an author at University of Chicago specializing in medicine, cell biology, and neuroscience.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	8	8	340
2	Social Sciences	5	5	172
3	Health Sciences	1	1	18
4	Natural Sciences	1	1	119
5	Biochemistry	1	1	68

Virginie Buggia-Prévot

Publications

1.5K

Citations

H-Index

Page 1

	Year	Citations
Presynaptic dystrophic neurites surrounding amyloid plaques are sites of microtubule disruption, BACE1 elevation, and increased Aβ generation in Alzheimer’s disease Katherine R. Sadleir, Patty C. Kandalepas, Virginie Buggia-Prévot, Acta Neuropathologica	2016	269
Axonal BACE1 dynamics and targeting in hippocampal neurons: a role for Rab11 GTPase Virginie Buggia-Prévot, Celia Fernandez, Sean M. Riordan, Molecular Neurodegeneration	2014	192
Predominant expression of Alzheimer’s disease-associated BIN1 in mature oligodendrocytes and localization to white matter tracts Pierre De Rossi, Virginie Buggia-Prévot, Benjamin L.L. Clayton, Molecular Neurodegeneration	2016	156
A Paired RNAi and RabGAP Overexpression Screen Identifies Rab11 as a Regulator of β-Amyloid Production Vinod Udayar, Virginie Buggia-Prévot, Rita Guerreiro, Cell Reports	2013	150
NFκB-dependent Control of BACE1 Promoter Transactivation by Aβ42 Virginie Buggia-Prévot, Jean Sévalle, Frédéric Checler Journal of Biological Chemistry Molecular RegulationImmunologyAbeta DomainNeuroinflammationTranscriptional Regulation	2008	119
Nuclear Factor-κB Regulates βAPP and β- and γ-Secretases Differently at Physiological and Supraphysiological Aβ Concentrations L. Chami, Virginie Buggia-Prévot, Eric Duplan, Journal of Biological Chemistry	2012	113
Pathogenic tau accelerates aging-associated activation of transposable elements in the mouse central nervous system Paulino Ramirez, Gabrielle Zuniga, Wenyan Sun, Progress in Neurobiology	2021	76
A Function for EHD Family Proteins in Unidirectional Retrograde Dendritic Transport of BACE1 and Alzheimer’s Disease Aβ Production Virginie Buggia-Prévot, Celia Fernández-Sanz, Vinod Udayar, Cell Reports	2013	74
Lack of BACE1 S-palmitoylation reduces amyloid burden and mitigates memory deficits in transgenic mouse models of Alzheimer’s disease Robert J. Andrew, Celia Fernández-Sanz, Molly Stanley, Proceedings of the National Academy of Sciences Transgenic Mouse ModelsSynaptic TransmissionBace1 ExpressionAmyloid Precursor ProteinSynaptic Signaling	2017	68
Ca2+ Influx through Store-operated Ca2+ Channels Reduces Alzheimer Disease β-Amyloid Peptide Secretion William Zeiger, Kulandaivelu S. Vetrivel, Virginie Buggia-Prévot, Journal of Biological Chemistry Ca2+ InfluxProtein SecretionAlzheimer's DiseaseMolecular PhysiologyMolecular Neuroscience	2013	40

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