Kimberly A. McAllister

Author

National Institute of Environmental Health Sciences ORCID

Also Known As

Baccarelli, Andrea A., K A McAllister, K. A. McAllister, K. McAllister, K.A. McAllister, Kimberly A McAllister, Kimberly A. McAllister, Kimberly A. Mcallister, Kimberly Ann McAllister, Kimberly McAllister

Publications

3.8K

Citations

H-Index

Concepts

All Affiliations

National Institute of Environmental Health Sciences National Institutes of Health Duke Medical Center Duke University Hospital Duke University

About

Kimberly A. McAllister is an author at National Institute of Environmental Health Sciences specializing in medicine, genetics, and pathology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	16	17	2.7K
2	Public Health	7	7	604
3	Biology	2	2	82
4	Biostatistics	2	2	266
5	Health Sciences	1	1	110

Kimberly A. McAllister

Publications

3.1K

Citations

H-Index

Page 1

	Year	Citations
Endoglin, a TGF-β binding protein of endothelial cells, is the gene for hereditary haemorrhagic telangiectasia type 1 Kimberly A. McAllister, K.M. Grogg, David W. Johnson, Nature Genetics Endothelial Cell PathobiologyVascular DiseaseDevelopmental BiologyAngiogenesisEndothelial Cells	1994	1.5K
Current Challenges and New Opportunities for Gene-Environment Interaction Studies of Complex Diseases Kimberly A. McAllister, Leah E. Mechanic, Christopher I. Amos, American Journal of Epidemiology Environmental Exposure AssessmentGeneticsGenetic EpidemiologyMolecular BiologyMultiomics	2017	211
A second locus for hereditary hemorrhagic telangiectasia maps to chromosome 12. David W. Johnson, Jonathan Berg, Carol J. Gallione, Genome Research GeneticsGenetic EpidemiologyHuman PolymorphismPathologyMolecular Genetics	1995	176
Cancer susceptibility of mice with a homozygous deletion in the COOH-terminal domain of the Brca2 gene. Kimberly A. McAllister, L. Michelle Bennett, Chris D. Houle, PubMed PathologyCancer BiologyEpigeneticsTumor BiologyCooh-terminal Domain	2002	118
Gene‐environment interplay in common complex diseases: forging an integrative model—recommendations from an NIH workshop Ebony Bookman, Kimberly A. McAllister, Elizabeth M. Gillanders, Genetic Epidemiology	2011	117
Six novel mutations in the endoglin gene in hereditary hemorrhagic telangiectasia type 1 suggest a dominant-negative effect of receptor function Kimberly A. McAllister, Melanie A. Baldwin, Arun K. Thukkani, Human Molecular Genetics Endoglin GeneGeneticsPathologyMolecular GeneticsDisease Gene Identification	1995	110
The NIEHS TaRGET II Consortium and environmental epigenomics Ting Wang, Erica C. Pehrsson, Deepak Purushotham, Nature Biotechnology BioinformaticsOmics DatasetsGeneticsComputational GenomicsEpigenomics	2018	106
Genetic heterogeneity in hereditary haemorrhagic telangiectasia: possible correlation with clinical phenotype. Kimberly A. McAllister, Felicia Lennon, B Bowles-Biesecker, Journal of Medical Genetics	1994	103
Hematopoietic Dysfunction in a Mouse Model for Fanconi Anemia Group D1 Susana Navarro, Néstor W. Meza, Óscar Quintana-Bustamante, Molecular Therapy	2006	102
Mutation and expression analysis of the endoglin gene in hereditary hemorrhagic telangiectasia reveals null alleles Carol J. Gallione, Daniel J. Klaus, Eric Y. Yeh, Human Mutation Expression AnalysisHht KindredsMendelian DisorderHereditary Hemorrhagic TelangiectasiaGenetic Disorder	1998	100

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