Mark S. Forman - Concepedia

Author

Mark S. Forman

Also Known As

Forman, Mark, Forman, Mark S, M FORMAN, M Forman, M S Forman, M. Forman, M. S. Forman, M.S. Forman, MARK S FORMAN, Mark Forman

120

Publications

13K

Citations

H-Index

Concepts

All Affiliations

University of Pennsylvania Institute on Aging Merck & Co., Inc., Rahway, NJ, USA (United States)Washington University in St. Louis Ludwig-Maximilians-Universität München

About

Mark S. Forman is an author at University of Pennsylvania specializing in medicine, neuroscience, and alzheimer's disease.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	26	31	5.7K
2	Social Sciences	10	10	2.5K
3	Natural Sciences	3	3	825
4	Biochemistry	2	2	233
5	Arts	1	1	25

Mark S. Forman

Publications

11.5K

Citations

H-Index

Page 1

	Year	Citations
Pathological TDP‐43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with <i>SOD1</i> mutations Ian R. Mackenzie, Eileen H. Bigio, Paul G. Ince, Annals of Neurology	2007	975
Initiation and Synergistic Fibrillization of Tau and Alpha-Synuclein Benoit I. Giasson, Mark S. Forman, Makoto Higuchi, Science	2003	869
Phosphorylation of S409/410 of TDP-43 is a consistent feature in all sporadic and familial forms of TDP-43 proteinopathies Manuela Neumann, Linda K. Kwong, Edward B. Lee, Acta Neuropathologica Signal TransductionMedicineGeneticsTdp-43 ProteinopathiesNatural Sciences	2009	592
Role for Akt3/Protein Kinase Bγ in Attainment of Normal Brain Size Rachael Easton, Han Cho, Kristin Roovers, Molecular and Cellular Biology Brain DevelopmentBrain ScienceMetabolic RemodelingNormal Organ SizeSynaptic Signaling	2005	547
TDP-43 in Familial and Sporadic Frontotemporal Lobar Degeneration with Ubiquitin Inclusions Nigel J. Cairns, Manuela Neumann, Eileen H. Bigio, American Journal Of Pathology Ubiquitin InclusionsMendelian DisorderGenetic DisorderGeneticsInherited Metabolic Disease	2007	525
Frontotemporal dementia: Clinicopathological correlations Mark S. Forman, Jennifer Farmer, Julene K. Johnson, Annals of Neurology	2006	476
Alzheimer's Aβ(1–42) is generated in the endoplasmic reticulum/intermediate compartment of NT2N cells David G. Cook, Mark S. Forman, Jane C. Sung, Nature Medicine Neurodegenerative DiseasesMolecular NeuroscienceAlzheimer's DiseaseNt2n CellsSynaptic Transmission	1997	441
Frontotemporal Lobar Degeneration Julene K. Johnson, Janine Diehl, Mario F. Mendez, Archives of Neurology	2005	413
The BACE1 inhibitor verubecestat (MK-8931) reduces CNS β-amyloid in animal models and in Alzheimer’s disease patients Matthew Kennedy, Andrew W. Stamford, Xia Chen, Science Translational Medicine Glucose HomeostasisBace1 Inhibitor VerubecestatEnzyme 1Social SciencesNeuroinflammation	2016	407
Concomitant TAR-DNA-Binding Protein 43 Pathology Is Present in Alzheimer Disease and Corticobasal Degeneration but Not in Other Tauopathies Kunihiro Uryu, Hanae Nakashima‐Yasuda, Mark S. Forman, Journal of Neuropathology & Experimental Neurology Corticobasal DegenerationGeneticsMolecular BiologyPathologyNeurochemical Biomarkers	2008	381

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