Kristen E. Mengwasser

Author

Brigham and Women's Hospital

Also Known As

K.E. Mengwasser, Kristen E. Mengwasser, Kristen Mengwasser, Kristen E. Mengwasser, Mengwasser, Kristen

Publications

3.3K

Citations

H-Index

Concepts

All Affiliations

Brigham and Women's Hospital Harvard University Howard Hughes Medical Institute Dana-Farber Cancer Institute Broad Institute

About

Kristen E. Mengwasser is an author at Brigham and Women's Hospital specializing in medicine, cell biology, and systems biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	4	4	3.1K
2	Cell Biology	3	3	2.3K
3	Natural Sciences	1	1	2.1K
4	Molecular Biology	1	1	2.1K
5	Molecular Genetics	1	1	194

Kristen E. Mengwasser

Publications

3.3K

Citations

H-Index

Page 1

	Year	Citations
The T790M mutation in EGFR kinase causes drug resistance by increasing the affinity for ATP Cai‐Hong Yun, Kristen E. Mengwasser, Angela V. Toms, Proceedings of the National Academy of Sciences Atp Binding PocketMolecular BiologyT790m MutationTumor BiologyDrug Resistance	2008	2.1K
Cumulative Haploinsufficiency and Triplosensitivity Drive Aneuploidy Patterns and Shape the Cancer Genome Teresa Davoli, Andrew Wei Xu, Kristen E. Mengwasser, Cell ChromatinGenome InstabilityCumulative HaploinsufficiencySomatic VariantTumor Heterogeneity	2013	873
Genetic Screens Reveal FEN1 and APEX2 as BRCA2 Synthetic Lethal Targets Kristen E. Mengwasser, Richard O. Adeyemi, Yumei Leng, Molecular Cell GeneticsSynthetic BiologyGenetic MechanismMolecular GeneticsMedicine	2019	194
Identification of FUBP1 as a Long Tail Cancer Driver and Widespread Regulator of Tumor Suppressor and Oncogene Alternative Splicing Jessica S. Elman, Thomas K. Ni, Kristen E. Mengwasser, Cell Reports	2019	46
Hirudin Binding Reveals Key Determinants of Thrombin Allostery Kristen E. Mengwasser, Leslie A. Bush, Peter Shih, Journal of Biological Chemistry	2005	36
Genetic analysis of cancer drivers reveals cohesin and CTCF as suppressors of PD-L1 Ena Oreskovic, Emily C. Wheeler, Kristen E. Mengwasser, Proceedings of the National Academy of Sciences	2022	33
Functional genomics reveals that tumors with activating phosphoinositide 3-kinase mutations are dependent on accelerated protein turnover Teresa Davoli, Kristen E. Mengwasser, Jingjing Duan, Genes & Development Tumor BiologyPhosphoinositide 3-Kinase MutationsOncologyReceptor Tyrosine KinaseRibosomal Protein Translation	2016	12

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