Thomas Koed Doktor - Concepedia

Author

Thomas Koed Doktor

Also Known As

Doktor, T, Doktor, T. K., Doktor, Thomas, E. S. Jul, Thomas Koed Doktor, Thomas Doktor, Thomas K Doktor, Thomas K. Doktor, Thomas Koed Doktor

Publications

1.7K

Citations

H-Index

Concepts

All Affiliations

University of Southern Denmark Aarhus University Aarhus University Hospital Cold Spring Harbor Laboratory Charles University

About

Thomas Koed Doktor is an author at University of Southern Denmark specializing in medicine, molecular genetics, and genetics.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	14	14	994
2	Natural Sciences	7	7	386
3	Biochemistry	4	4	102
4	Engineering	3	3	59
5	Biology	1	1	210

Thomas Koed Doktor

Publications

1.6K

Citations

H-Index

Page 1

	Year	Citations
Tissue-resident macrophages in omentum promote metastatic spread of ovarian cancer Anders Etzerodt, Morgane Moulin, Thomas Koed Doktor, The Journal of Experimental Medicine MedicineTumor GrowthImmunologyTissue-resident MacrophagesGynecology	2020	333
Splicing factor 1 modulates dietary restriction and TORC1 pathway longevity in C. elegans Caroline Heintz, Thomas Koed Doktor, Anne Lanjuin, Nature BiologyLifespan ExtensionTranscriptional RegulationDevelopmental BiologyNatural Sciences	2016	210
CUGBP1 and MBNL1 preferentially bind to 3′ UTRs and facilitate mRNA decay Akio Masuda, Henriette S. Andersen, Thomas Koed Doktor, Scientific Reports	2012	176
Seemingly Neutral Polymorphic Variants May Confer Immunity to Splicing-Inactivating Mutations: A Synonymous SNP in Exon 5 of MCAD Protects from Deleterious Mutations in a Flanking Exonic Splicing Enhancer Karsten Nielsen, Suzette Sørensen, Luca Cartegni, The American Journal of Human Genetics Synonymous SnpAllelic VariantGeneticsRna SplicingMolecular Biology	2007	148
RNA-sequencing of a mouse-model of spinal muscular atrophy reveals tissue-wide changes in splicing of U12-dependent introns Thomas Koed Doktor, Yimin Hua, Henriette S. Andersen, Nucleic Acids Research	2016	104
DeepCLIP: predicting the effect of mutations on protein–RNA binding with deep learning Alexander G. B. Grønning, Thomas Koed Doktor, Simon J. Larsen, Nucleic Acids Research	2020	101
Global identification of hnRNP A1 binding sites for SSO-based splicing modulation Gitte Hoffmann Bruun, Thomas Koed Doktor, Jonas Borch-Jensen, BMC Biology	2016	73
DeepCLIP: Predicting the effect of mutations on protein-RNA binding with Deep Learning Alexander G. B. Grønning, Thomas Koed Doktor, Simon J. Larsen,	2019	59
The deep intronic c.903+469T>C mutation in the <i>MTRR</i> gene creates an SF2/ASF binding exonic splicing enhancer, which leads to pseudoexon activation and causes the cblE type of homocystinuria Katerina Homolova, Petra Zavadáková, Thomas Koed Doktor, Human Mutation C MutationDeep Intronic C.903+469tGeneticsRna SplicingGenomic Mechanism	2010	56
The prevalent deep intronic c. 639 + 919 G > A GLA mutation causes pseudoexon activation and Fabry disease by abolishing the binding of hnRNPA1 and hnRNP A2/B1 to a splicing silencer Bruno Palhais, Maja Dembić, Rugivan Sabaratnam, Molecular Genetics and Metabolism Transcriptional RegulationFabry DiseaseGenetic DisorderGeneticsPseudoexon Activation	2016	48

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