Simon N. Willis - Concepedia

Author

Simon N. Willis

Walter and Eliza Hall Institute of Medical Research ORCID

Also Known As

S N Willis, S. N. Willis, S. WILLIS, S. Willis, S.N. Willis, Simon N Willis, Simon N. Willis, Simon Willis

Publications

9.4K

Citations

H-Index

Concepts

All Affiliations

Walter and Eliza Hall Institute of Medical Research The University of Melbourne Harvard University Yale University Brigham and Women's Hospital

About

Simon N. Willis is an author at Walter and Eliza Hall Institute of Medical Research specializing in immunology, medicine, and cell biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	16	16	5.6K
2	Immunology	16	16	5.6K
3	Cell Biology	11	11	5.2K
4	Allergy	2	2	183
5	Cellular Physiology	1	1	77

Simon N. Willis

Publications

8.3K

Citations

H-Index

Page 1

	Year	Citations
Differential Targeting of Prosurvival Bcl-2 Proteins by Their BH3-Only Ligands Allows Complementary Apoptotic Function Lin Chen, Simon N. Willis, Andrew H. Wei, Molecular Cell MedicineApoptosisImmunologyDifferential TargetingCell Death	2005	1.8K
The BH3 mimetic ABT-737 targets selective Bcl-2 proteins and efficiently induces apoptosis via Bak/Bax if Mcl-1 is neutralized Mark F. van Delft, Andrew H. Wei, Kylie D. Mason, Cancer Cell ApoptosisImmunologyCell DeathAnti-cancer AgentMedicine	2006	1.2K
Proapoptotic Bak is sequestered by Mcl-1 and Bcl-x <sub>L</sub> , but not Bcl-2, until displaced by BH3-only proteins Simon N. Willis, Chen Lin, Grant Dewson, Genes & Development	2005	1.2K
Apoptosis Initiated When BH3 Ligands Engage Multiple Bcl-2 Homologs, Not Bax or Bak Simon N. Willis, Jamie I. Fletcher, Thomas Kaufmann, Science Pro-survival Bcl-2-like RelativesSignal TransductionBh3-only MembersSignaling PathwayCell Regulation	2007	1.1K
Transcriptional profiling of mouse B cell terminal differentiation defines a signature for antibody-secreting plasma cells Wei Shi, Yang Liao, Simon N. Willis, Nature Immunology Transcriptional ProfilingAutoimmune DiseaseLymphocyte DevelopmentImmune Cell DevelopmentImmunology	2015	377
Polθ inhibitors elicit BRCA-gene synthetic lethality and target PARP inhibitor resistance Diana Zatreanu, Helen M.R. Robinson, Omar Alkhatib, Nature Communications	2021	336
Epstein–Barr virus infection is not a characteristic feature of multiple sclerosis brain Simon N. Willis, Christine Stadelmann, Scott J. Rodig, Brain	2009	274
Interleukin 15–mediated survival of natural killer cells is determined by interactions among Bim, Noxa and Mcl-1 Nicholas D. Huntington, Hamsa Puthalakath, Priscilla Gunn, Nature Immunology Interleukin 15–MediatedInnate Immune SystemImmunologyImmune RegulationCell Death	2007	257
Related B cell clones populate the meninges and parenchyma of patients with multiple sclerosis Laura Lovato, Simon N. Willis, Scott J. Rodig, Brain B Cell ClonesNeurological DisorderImmunologyNeurological ProgressPathology	2011	222
A novel BH3 ligand that selectively targets Mcl-1 reveals that apoptosis can proceed without Mcl-1 degradation Erinna F. Lee, Peter E. Czabotar, Mark F. van Delft, The Journal of Cell Biology	2008	169

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