José Miguel Escandell

Author

Also Known As

J Escandell, J. Escandell, Jose Escandell, Jose M Escandell, Jose M. Escandell, Jose Miguel Escandell, José Escandell, José M. Escandell, José Miguel Escandell

Publications

1.9K

Citations

H-Index

Concepts

All Affiliations

Universitat de València CRUK/MRC Oxford Institute for Radiation Oncology Spanish National Cancer Research Centre The Netherlands Cancer Institute Instituto Gulbenkian de Ciência

About

José Miguel Escandell is an author at Universitat de València specializing in medicine, cell biology, and genome instability.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	7	7	458
2	Cell Biology	5	5	392
3	Natural Sciences	2	2	250
4	Allergy	1	1	37
5	Immunology	1	1	37

José Miguel Escandell

Publications

774

Citations

H-Index

Page 1

	Year	Citations
BRCA2 acts as a RAD51 loader to facilitate telomere replication and capping Sophie Badie, José Miguel Escandell, Peter Bouwman, Nature Structural & Molecular Biology ChromatinGenome InstabilityNatural SciencesDna ReplicationMolecular Biology	2010	156
FoxM1 repression during human aging leads to mitotic decline and aneuploidy-driven full senescence Joana Catarina Macedo, Sara Vaz, Björn Bakker, Nature Communications	2018	130
ATM/ATR checkpoint activation downregulates CDC25C to prevent mitotic entry with uncapped telomeres Maria Thanasoula, José Miguel Escandell, Natsuko Suwaki, The EMBO Journal Cell DivisionNatural SciencesDna ReplicationMolecular BiologyMitotic Entry	2012	94
Cucurbitacin R Reduces the Inflammation and Bone Damage Associated with Adjuvant Arthritis in Lewis Rats by Suppression of Tumor Necrosis Factor-α in T Lymphocytes and Macrophages José Miguel Escandell, M. Carmen Recio, Salvador Máñez, Journal of Pharmacology and Experimental Therapeutics	2006	68
Activated kRas protects colon cancer cells from cucurbitacin-induced apoptosis: The role of p53 and p21 José Miguel Escandell, Pawan Kaler, M. Carmen Recio, Biochemical Pharmacology Cucurbitacin-induced ApoptosisCancer ResearchOncologyMedicineApoptosis	2008	52
ARF triggers senescence in Brca2-deficient cells by altering the spectrum of p53 transcriptional targets Ana Rita Carlos, José Miguel Escandell, Panagiotis Kotsantis, Nature Communications Genome InstabilityCell RegulationBrca2-deficient CellsCellular SenescenceArf Triggers Senescence	2013	45
p53 Prevents Entry into Mitosis with Uncapped Telomeres Maria Thanasoula, José Miguel Escandell, Paula Martínez, Current Biology Genome InstabilityCell DivisionP53 Prevents EntryCell DeathMedicine	2010	45
New insight into the inhibition of the inflammatory response to experimental delayed-type hypersensitivity reactions in mice by scropolioside A Esperanza Bas, M. Carmen Recio, Salvador Máñez, European Journal of Pharmacology InflammationCytokineScropolioside AAnti-inflammatoryAllergy	2006	37
The fission yeast Stn1-Ten1 complex limits telomerase activity via its SUMO-interacting motif and promotes telomeres replication Samah Matmati, Mélina Vaurs, José Miguel Escandell, Science Advances	2018	33
Dihydrocucurbitacin B Inhibits Delayed Type Hypersensitivity Reactions by Suppressing Lymphocyte Proliferation José Miguel Escandell, M. Carmen Recio, Salvador Máñez, Journal of Pharmacology and Experimental Therapeutics	2007	30

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