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Dimethyl fumarate improves cognitive deficits in chronic cerebral hypoperfusion rats by alleviating inflammation, oxidative stress, and ferroptosis via NRF2/ARE/NF-κB signal pathway

Cerebral Vascular RegulationNeurodegenerative DiseasesNeurobiology Of DiseaseCognitive PerformanceMedicineDimethyl Fumarate ImprovesBrain InjuryNeurologyNeuroscienceNeuroprotectionCerebral Blood FlowNrf2/are/nf-κb Signal PathwayPharmacologySocial SciencesOxidative StressNeuroinflammation