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Mechanical stiffness augments ligand-dependent progesterone receptor B activation via MEK 1/2 and Rho/ROCK–dependent signaling pathways in uterine fibroid cells

GynecologyMechanotransductionCellular PhysiologyMatrix BiologyPublic HealthMyometrial ContractilityCell SignalingMolecular SignalingMechanobiologyUterine FibroidsMolecular PhysiologyMek 1/2MechanosensingCell BiomechanicsCell BiologyUterine Fibroid CellsUterine ReceptivityCell-matrix InteractionMedicineExtracellular Matrix