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Interleukin-17 upregulation participates in the pathogenesis of heart failure in mice via NF-κB-dependent suppression of SERCA2a and Cav1.2 expression

InflammationCytokineHeart FailureMolecular PhysiologyDisease PathophysiologyCav1.2 ExpressionPhysiologyImmunologyChronic InflammationVascular BiologyImmune MediatorMedicineCell BiologyCell SignalingNf-κb-dependent SuppressionImmune Dysregulation