Richard S. Lewis - Concepedia

Author

Richard S. Lewis

Also Known As

Lewis, R.S., Lewis, Richard S., R S Lewis, R. Lewis, R. S. Lewis, R.S. Lewis, Richard Lewis, Richard S Lewis, Richard S. Lewis, Richard S. Lewis

140

Publications

25.4K

Citations

H-Index

Concepts

All Affiliations

Stanford University University of California, Irvine Howard Hughes Medical Institute Harvard University University of California, San Francisco

About

Richard S. Lewis is an author at Stanford University specializing in medicine, cellular physiology, and signal transduction.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	29	31	14.1K
2	Natural Sciences	9	9	3.1K
3	Biochemistry	6	6	1.3K
4	Social Sciences	3	3	1K
5	Engineering	2	2	263

Richard S. Lewis

Publications

20.3K

Citations

H-Index

Page 1

	Year	Citations
A mutation in Orai1 causes immune deficiency by abrogating CRAC channel function Stefan Feske, Yousang Gwack, Murali Prakriya, Nature Crac Channel FunctionImmunogeneticsMolecular ImmunologyImmune DeficiencyAutoimmune Disease	2006	2.2K
Calcium oscillations increase the efficiency and specificity of gene expression Ricardo E. Dolmetsch, Keli Xu, Richard S. Lewis Nature Calcium OscillationsDevelopmental BiologySignal TransductionGeneticsGene Expression	1998	1.9K
Differential activation of transcription factors induced by Ca2+ response amplitude and duration Ricardo E. Dolmetsch, Richard S. Lewis, Christopher C. Goodnow, Nature Transcription FactorsSignal TransductionCa2+ Response AmplitudeSignaling PathwayMolecular Regulation	1997	1.7K
Differential activation of transcription factors induced by Ca2+ response amplitude and duration Ricardo E. Dolmetsch, Richard S. Lewis, Christopher C. Goodnow, Nature Transcription FactorsSignal TransductionCa2+ Response AmplitudeSignaling PathwayMolecular Regulation	1997	1.5K
STIM1 Clusters and Activates CRAC Channels via Direct Binding of a Cytosolic Domain to Orai1 Chan Young Park, Paul Hoover, Franklin M. Mullins, Cell Activates Crac ChannelsSignal TransductionMolecular PhysiologyMedicineCytosolic Domain	2009	938
Ca2+ store depletion causes STIM1 to accumulate in ER regions closely associated with the plasma membrane Minnie M. Wu, JoAnn Buchanan, Riina M. Luik, The Journal of Cell Biology Stim1 RedistributionStim1 PunctaMolecular BiologyCytoskeletonCellular Physiology	2006	768
Mitogen-regulated Ca2+ current of T lymphocytes is activated by depletion of intracellular Ca2+ stores. Adam Zweifach, Richard S. Lewis Proceedings of the National Academy of Sciences	1993	750
Abnormal Calcium Handling Properties Underlie Familial Hypertrophic Cardiomyopathy Pathology in Patient-Specific Induced Pluripotent Stem Cells Feng Lan, Andrew S. Lee, Ping Liang, Cell stem cell Cardiac MuscleCardiomyopathyHeart FailureDevelopmental BiologyInduced Pluripotent Stem Cell	2013	666
The elementary unit of store-operated Ca2+ entry: local activation of CRAC channels by STIM1 at ER–plasma membrane junctions Riina M. Luik, Minnie M. Wu, JoAnn Buchanan, The Journal of Cell Biology	2006	636
Role of Membrane Potential and Hydrogen Bonding in the Mechanism of Translocation of Guanidinium-Rich Peptides into Cells Jonathan B. Rothbard, Theodore C. Jessop, Richard S. Lewis, Journal of the American Chemical Society	2004	603

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