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Sulforaphane ameliorates ethanol plus carbon tetrachloride-induced liver fibrosis in mice through the Nrf2-mediated antioxidant response and acetaldehyde metabolization with inhibition of the LPS/TLR4 signaling pathway

InflammationAcetaldehyde MetabolizationHepatologyBiochemistryMedicineLiver PhysiologyLipid PeroxidationNrf2-mediated Antioxidant ResponseHepatotoxicityAlcohol-related Liver DiseaseHepatology FibrosisMetabolomicsReactive Oxygen SpeciePharmacologyRedox BiologyDrug-induced Liver InjuryOxidative Stress