Exosomes from EV71‐infected oral epithelial cells can transfer miR‐30a to promote EV71 infection

Abstract

Exosomes released from EV71-infected OE cells selectively packaged high level of miR-30a that can be functionally transferred to the recipient macrophages resulted in targeting MyD88 and subsequently inhibited type I interferon production in receipt cells, thus promoting the EV71 replication.