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Dosing depending on SIRT3 activity attenuates doxorubicin-induced cardiotoxicity via elevated tolerance against mitochondrial dysfunction and oxidative stress

InflammationMitochondrial DysfunctionMedicineLipid PeroxidationPhysiologyCell DeathReactive Oxygen SpecieSirt3 ActivityCardiovascular ToxicityPharmacologyRedox BiologyOxidative Stress