The effect of <scp>NLRP</scp> inflammasome on the regulation of <scp>AGE</scp>s‐induced inflammatory response in human periodontal ligament cells

Abstract

Advanced glycation end products induced inflammatory response in HPDLCs via NLRP1-inflammasome and NLRP3-inflammasome activation in which NF-κB signal pathway was involved. Besides, AGEs promoted the inflammatory response of MDP via NOD2, NLRP1-inflammasome, and NLRP3-inflammasome.