Cell Reports · 2017 · 86 citations · 52 references
Life SciencesNeurobiologyNeuroscienceMolecular NeurobiologyMolecular NeuroscienceSynaptic PlasticityGlutamatergic SignalingLong-term PotentiationNeurotransmissionReceptor Signaling In NeuronsSynaptic Strength RegulationSynaptic TransmissionCalcium-dependent PlasticityCalcium-dependent Protein Kinase IiCalmodulin-dependent Protein Kinase IiLong-term Synaptic PlasticityNeuronal Cell Death PathwaysNmda Receptor SignalingReceptor AutophosphorylationSynaptic Plasticity Signaling
/CaM results in synaptic DAPK1 removal during LTP but retention during LTD. A pharmacogenetic approach showed that suppression of CaMKII/GluN2B binding is a DAPK1 function required for LTD.
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