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Nitric oxide (NO)-mediated mitochondrial damage plays a critical role in T-2 toxin-induced apoptosis and growth hormone deficiency in rat anterior pituitary GH3 cells

Reductive StressMitochondrial DamageGrowth HormoneMitochondrial FunctionHuman GrowthNitric OxideT-2 Toxin-induced ApoptosisPhysiologyReactive Nitrogen SpecieCell DeathMetabolismEndocrinologyMedicineCell BiologyNitrosative StressOxidative Stress