Publication | Open Access
CaMKIIδ subtypes differentially regulate infarct formation following ex vivo myocardial ischemia/reperfusion through NF-κB and TNF-α
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Citations
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References
2017
Year
InflammationMolecular PhysiologyCardiovascular DiseaseCell SignalingImmunologyVascular BiologyCell BiologyRegulate Infarct FormationMedicineCardiovascular Disease PathogenesisCardiologyAcute Myocardial InfarctionMyocardial Infarction
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