Publication | Open Access
Targeting the histone methyltransferase G9a activates imprinted genes and improves survival of a mouse model of Prader–Willi syndrome
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Citations
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References
2016
Year
Histone ModificationsEpigenetic ChangeGeneticsEpigeneticsTranscriptional RegulationMolecular EpigeneticsGerm Cell DevelopmentPrader–willi SyndromeMouse ModelHistone Methyltransferase G9aGene ExpressionEpigenetic RegulationChromatin FunctionChromatinDevelopmental BiologyChromatin StructureNatural SciencesEpigenomicsGene RegulationMedicineCell Development
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