Publication | Open Access
C66 ameliorates diabetic nephropathy in mice by both upregulating NRF2 function via increase in miR-200a and inhibiting miR-21
99
Citations
40
References
2016
Year
InflammationMetabolic SyndromeUpregulating Nrf2 FunctionMedicineDiabetesImmunologyDiabetic Kidney DiseaseInhibiting Mir-21Diabetic NephropathyMicrorna DetectionChronic Kidney DiseaseCell BiologyCell SignalingNephrologyKidney Research
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