Contribution of <i>KRAS</i> mutations and c.2369C > T (p.T790M) <i>EGFR</i> to acquired resistance to EGFR-TKIs in <i>EGFR</i> mutant NSCLC: a study on circulating tumor DNA - Concepedia

Concepedia

Publication | Open Access

Contribution of <i>KRAS</i> mutations and c.2369C > T (p.T790M) <i>EGFR</i> to acquired resistance to EGFR-TKIs in <i>EGFR</i> mutant NSCLC: a study on circulating tumor DNA

DOI Full Paper Access

84

Citations

27

References

2016

Year

Marzia Del Re, Marcello Tiseo, Paola Bordi, Armida D’Incecco, Andrea Camerini, Iacopo Petrini, M Lucchesi, Alessandro Inno, Daniele Spada, Enrico Vasile,

Abstract

MUTKRAS could be an additional mechanism of escape from EGFR-TKI inhibition and cftDNA is a feasible approach to monitor the molecular development of drug resistance.

References

	Year	Citations

Page 1