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ATP leakage induces P2XR activation and contributes to acute synaptic excitotoxicity induced by soluble oligomers of β-amyloid peptide in hippocampal neurons

Synaptic PlasticityMolecular NeuroscienceSynaptic SignalingNeurophysiologyMedicineSynaptic TransmissionSoluble Oligomersβ-Amyloid PeptideSynaptic ExcitotoxicityProtein MisfoldingNeuroprotectionNeuroscienceMolecular NeurobiologyPharmacologyNeurochemistrySocial Sciences