Inhibition of renin angiotensin aldosterone system causes abrogation of obliterative airways disease through inhibition of tumor necrosis factor-α–dependant interleukin-17

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IL-6 programs TH-17 cell differentiation by promoting sequential engagement of the IL-21 and IL-23 pathways

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Angiotensin II-forming pathways in normal and failing human hearts.

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IL-17–dependent cellular immunity to collagen type V predisposes to obliterative bronchiolitis in human lung transplants

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Blocking angiotensin-converting enzyme induces potent regulatory T cells and modulates TH1- and TH17-mediated autoimmunity

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