Fumonisin <scp>FB</scp>1 treatment acts synergistically with methyl donor deficiency during rat pregnancy to produce alterations of <scp>H</scp>3‐ and <scp>H</scp>4‐histone methylation patterns in fetuses

Abstract

Considering that humans are exposed to FB1 levels above the PMTDI, this study is relevant because it suggests that low doses of FB1 interact with MDD thus contributing to disrupt the epigenetic landscape.