Publication | Open Access
Neutrophils produce interleukin-17B in rheumatoid synovial tissue
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Citations
32
References
2013
Year
IL-17B, which is present in synovium, may contribute to the pathogenesis of RA. IL-17B can enhance the effects of TNF-α on the production of cytokines and chemokines that control immune cell trafficking and neutrophil homeostasis in the inflamed tissues.
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