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Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or neutrophil infiltration

InflammationTraumatic Brain InjuryCell ApoptosisMedicinePharmacologyCell DeathBrain-immune InteractionBrain InjuryNeurologyNeuroprotectionNeuroscienceCerebral Blood FlowNeuropathologyNeuroimmunologyAttenuated Microglial ActivationNeuroinflammation