Pro-inflammatory cytokines and soluble receptors in response to acute psychosocial stress: Differential reactivity in bipolar disorder

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A mechanism converting psychosocial stress into mononuclear cell activation

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Tumor necrosis factor soluble receptors circulate during experimental and clinical inflammation and can protect against excessive tumor necrosis factor alpha in vitro and in vivo.

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Stabilization of the bioactivity of tumor necrosis factor by its soluble receptors.

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Increased Stress-Induced Inflammatory Responses in Male Patients With Major Depression and Increased Early Life Stress

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