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Poly(ethylene glycol)s generate complement activation products in human serum through increased alternative pathway turnover and a MASP-2-dependent process

InflammationAlternative Pathway TurnoverComplement SystemEthylene GlycolSignal TransductionAutoimmune DiseaseProtein ExpressionMedicineImmunologyAutoimmunityImmunosuppressionImmunopathologyCellular BiochemistryHuman SerumCell BiologyCell Signaling