Publication | Closed Access
A procyanidin trimer, C1, promotes NO production in rat aortic endothelial cells via both hyperpolarization and PI3K/Akt pathways
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Citations
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References
2012
Year
InflammationPi3k/akt PathwaysSignal TransductionAngiogenesisMolecular PhysiologyMedicinePhysiologyEndothelial DysfunctionProcyanidin TrimerVascular BiologyCardiovascular FunctionPharmacologyCell BiologyCell SignalingCellular PhysiologyOxidative Stress
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