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Suppression of ERCC1 and Rad51 expression through ERK1/2 inactivation is essential in emodin-mediated cytotoxicity in human non-small cell lung cancer cells

Oncogenic AgentMedicineCell DeathEmodin-mediated CytotoxicityCancer BiologyTumor SuppressorCancer GeneticsRadiation OncologyOncologyCell BiologyErk1/2 InactivationLung CancerCancer ResearchRad51 Expression