Loss of teratogenic response to 2,3,7,8‐tetrachlorodibenzo‐<i>p</i>‐dioxin (TCDD) in mice lacking the Ah (dioxin) receptor

Abstract

These results clearly show that the AhR is involved in the malformation of the palate and kidney in mouse embryos caused by TCDD and suggests that the mechanism of its involvement differs between the cleft palate and hydronephrosis.