Publication | Closed Access

Inhibition of fatty acid oxidation by etomoxir impairs NADPH production and increases reactive oxygen species resulting in ATP depletion and cell death in human glioblastoma cells

OxysterolReactive Oxygen SpeciesGliomaBiochemistryMedicineLipid PeroxidationCell DeathAtp DepletionMetabolic RemodelingFatty Acid OxidationReactive Oxygen SpecieMetabolomicsMetabolismPharmacologyCell BiologyRedox BiologyOxidative Stress