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Point mutation at the ATP binding site of EGF receptor abolishes protein-tyrosine kinase activity and alters cellular routing

Signal TransductionSignaling PathwayProtein-tyrosine Kinase ActivityReceptor Tyrosine KinaseReceptor (Biochemistry)Molecular BiologyPoint MutationEgf ReceptorMedicineCell BiologyCell SignalingCellular PhysiologyProtein Phosphorylation