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Prevention of autoimmune diabetes in NOD mice by troglitazone is associated with modulation of ICAM-1 expression on pancreatic islet cells and IFN-γ expression in splenic T cells

ImmunologyImmune RegulationImmunologic MechanismImmune SystemImmunotherapyInsulin SignalingInflammationTumor ImmunityIcam-1 ExpressionRegulatory T Cell BiologyAutoimmune DiseaseAutoimmunityT Cell ImmunitySelf-toleranceNod MiceCell BiologyDiabetesCellular Immune ResponseMedicineAutoimmune Diabetes