SNPs in the KCNJ11-ABCC8 gene locus are associated with type 2 diabetes and blood pressure levels in the Japanese population

References

Page 1

	Year	Citations
Statistical significance for genomewide studies John D. Storey, Robert Tibshirani Proceedings of the National Academy of Sciences GeneticsGenomicsHigh Throughput SequencingFalse Positive RateGenome-wide Association Studies	2003	9.9K
The Structure of Haplotype Blocks in the Human Genome Stacey Gabriel, S. F. Schaffner, Huy Nguyen, Science GeneticsGenetic EpidemiologyHuman PolymorphismGenomicsGenome-wide Association Study	2002	5.8K
Foundations of Epidemiology. Annals of Internal Medicine Epidemiologic ResearchComputational EpidemiologyPreventive MedicineClinical EpidemiologyMedical History	1977	1.2K
Large-Scale Association Studies of Variants in Genes Encoding the Pancreatic β-Cell KATP Channel Subunits Kir6.2 (<i>KCNJ11</i>) and SUR1 (<i>ABCC8</i>) Confirm That the <i>KCNJ11</i> E23K Variant Is Associated With Type 2 Diabetes Anna L. Gloyn, Michael N. Weedon, Katharine R. Owen, Diabetes	2003	753
Foundations of Epidemiology James E. Banta American Journal of Tropical Medicine and Hygiene American Health CommunityEpidemiologic ResearchHealth StudiesPreventive MedicineClinical Epidemiology	1981	560
ATP-sensitive K+ channels in the hypothalamus are essential for the maintenance of glucose homeostasis Takashi Miki, Birgit Liss, Kohtaro Minami, Nature Neuroscience Energy HomeostasisMolecular PhysiologySignal TransductionNeurophysiologyAtp-sensitive K+ Channels	2001	506
Kir6.2 is required for adaptation to stress Leonid V. Zingman, Denice M. Hodgson, Peter Bast, Proceedings of the National Academy of Sciences	2002	308
The E23K Variant of Kir6.2 Associates With Impaired Post-OGTT Serum Insulin Response and Increased Risk of Type 2 Diabetes Eva-Maria D. Nielsen, Lars Hansen, Bendix Carstensen, Diabetes	2003	289
Glucose-Sensing in Glucagon-Like Peptide-1-Secreting Cells Frank Reimann, Fiona M. Gribble Diabetes	2002	283
KIR6.2 Polymorphism Predisposes to Type 2 Diabetes by Inducing Overactivity of Pancreatic β-Cell ATP-Sensitive K+ Channels Christina Schwanstecher, Ulrike Meyer, Mathias Schwanstecher Diabetes	2002	247

Page 1