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Very-late-antigen-4 (VLA-4)-mediated brain invasion by neutrophils leads to interactions with microglia, increased ischemic injury and impaired behavior in experimental stroke

ImmunologyCerebrovascular DiseaseCerebral Vascular RegulationNeuroinflammationInflammationStrokeBrain InjuryNeurologyNeuroimmunologyIschemic SyndromeAutoimmunityBrain-immune InteractionCerebral Blood FlowExperimental StrokeIschemic StrokeNeuroscienceNeutrophils LeadsMedicineBrain Invasion