Publication | Open Access
Aspirin triggered-lipoxin A4 reduces the adhesion of human polymorphonuclear neutrophils to endothelial cells initiated by preeclamptic plasma
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References
2012
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InflammationPreeclamptic PlasmaAnti-inflammatoryEndothelial CellsGranulocyteMedicineImmunologyHematologyBlood CellEndothelial DysfunctionVascular BiologyAspirin Triggered-lipoxin A4PharmacologyAtherosclerosis
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A series of prostaglandin F2-like compounds are produced in vivo in humans by a non-cyclooxygenase, free radical-catalyzed mechanism. Jason D. Morrow, Kristina E. Hill, Raymond F. Burk, Proceedings of the National Academy of Sciences Lipid PeroxidationSecondary MetaboliteRedox BiologyFree Radical-catalyzed MechanismOxidative Stress | 1990 | 1.9K |
2001 | 1.4K | |
1994 | 906 | |
2012 | 836 | |
1998 | 831 | |
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2006 | 565 |
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