Publication | Open Access

Keratitis-Ichthyosis-Deafness Syndrome-Associated Cx26 Mutants Produce Nonfunctional Gap Junctions but Hyperactive Hemichannels When Co-Expressed With Wild Type Cx43

Skin DevelopmentSignal TransductionWild Type Cx43MedicinePathogenesisMolecular BiologyCochlear DevelopmentHyperactive HemichannelsCell BiologyCell SignalingCell Development