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Intravenous Administration of Stable-Labeled N-Acetylcysteine Demonstrates an Indirect Mechanism for Boosting Glutathione and Improving Redox Status

Reductive StressIntravenous AdministrationBiochemistryRedox RegulatorMedicineLipid PeroxidationBoosting GlutathioneReactive Oxygen SpecieIndirect MechanismPharmacologyRedox BiologyOxidative Stress