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Toll-like receptor 5 deficiency attenuates interstitial cardiac fibrosis and dysfunction induced by pressure overload by inhibiting inflammation and the endothelial–mesenchymal transition

InflammationFibrosisInterstitial Cardiac FibrosisDisease PathophysiologyPhysiologyImmunologyToll-like Receptor 5Chronic InflammationEndothelial DysfunctionVascular BiologyPressure OverloadCardiovascular FunctionMedicineCell BiologyCell Signaling