<scp>GluN2C</scp>/<scp>GluN2D</scp> subunit‐selective <scp>NMDA</scp> receptor potentiator <scp>CIQ</scp> reverses <scp>MK</scp>‐801‐induced impairment in prepulse inhibition and working memory in <scp>Y</scp>‐maze test in mice

Abstract

Together, these results suggest that facilitation of GluN2C/GluN2D-containing receptors may serve as an important therapeutic strategy for treating positive and cognitive symptoms in schizophrenia.